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Gested that the consequences of S1P stimulation of endothelial cells had been mediated by activation from the VEGF receptor [75]. This S1P-mediated activation of VEGF was dependent on G i protein and Src family kinases. Furthermore, VEGF can stimulate an increase of S1P receptors, thereby enhancing the potential of S1P to activate VEGF receptors [76]. Lately, it has been demonstrated that S1P can


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